Home Endocrine Regulations 2011 Endocrine Regulations Vol.45, No.3, p.163-172, 2011

Journal info

Quarterly, 50 pp. per issue 
Founded: 1967
ISSN 1210-0668
E-ISSN 1336-0329

Published in English

Aims and Scope
Editorial Info
Submission Guidelines

Select Journal

Webshop Cart

Your Cart is currently empty.

Info: Your browser does not accept cookies. To put products into your cart and purchase them you need to enable cookies.

Endocrine Regulations Vol.45, No.3, p.163-172, 2011

Title: Brain-liver interactions during liver ischemia reperfusion injury: a minireview
Author: J. Bundzikova, Z. Pirnik, L. Lackovicova, B. Mravec, A. Kiss

Abstract: The liver is a vital organ, with a wide range of functions. This organ plays an important role in the metabolism, including the glycogen storage, decomposition of red blood cells, plasma protein synthesis, hormone production, and detoxification. The liver is innervated by sympathetic and parasympathetic nerves which are involved in the regulation of the hepatic metabolism. Tissue injury connected with ischemia and reperfusion has been implicated in several clinical settings, including myocardial infarction, brain ischemia, and organ transplantation. Consequences of the liver ischemia reperfusion injury (LIRI) induce first of all an organ failure and afterwards multiorgan system damages that may eventually lead to a death. Many models with an attempt to reduce harmful consequences of the LIRI, directing to develop a variety of prophylactic strategies, has been introduced including models of warm, cold or normothermic ischemia, ischemic pre- and post-conditionings, pharmacological interventions, etc. In spite of the improvements in the medical care and accumulation of a large amount of experimental data concerning the prevention of ischemia and reperfusion related injuries, many destructive processes explanation still remains problematic.

Year: 2011, Volume: 45, Issue: 3 Page From: 163, Page To: 172
Price: 20.00 €

© AEPress s.r.o
Copyright notice: For any permission to reproduce, archive or otherwise use the documents in the ELiS, please contact AEP.